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[RC] HR recovery - Ridecamp Guest

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Authors A Crisafulli, C Carta, F Melis, F Tocco, F Frongia, UM Santoboni, P 
Pagliaro, A Concu
Title   Haemodynamic responses following intermittent supramaximal exercise in 
athletes
Full source     Experimental Physiology, 2004, Vol 89, Iss 6, pp 665-674

We aimed to investigate haemodynamics during active and passive recovery 
following repeated bouts of supramaximal exercise. Seven male athletes 
underwent two sessions of supramaximal exercise which consisted of a warm-up 
and of five bouts of cycling at the maximum speed possible for 30 s against a 
resistance equivalent to 150% of the maximum workload achieved in a previous 
incremental test. Bouts were separated by 1 min of recovery and followed by 10 
min of recovery which was either active (pedalling at 40 W) or passive 
(completely rest seated on the cycle). Haemodynamic variables were evaluated by 
means of impedance cardiography. Heart rate (HR), stroke volume (SV), cardiac 
output (CO), mean blood pressure (MBP), thoracic electrical impedance (Z(0)) as 
an inverse index of central blood volume, and systemic vascular resistance 
(SVR) were assessed. The main findings were that active recovery, with respect 
to passive recovery, induced higher changes from baseline in HR (29.1 +/- 4.5 
versus +15.6 +/- 2.9 beats min(-1) at the 10th minute of recovery, P < 0.05), 
SV (+19.9 &PLUSMN; 5.6 versus -6.4 &PLUSMN; 3.3 ml, P < 0.01) and CO (+3.8 +/- 
1.2 versus +0.4 +/- 0.21 min(-1), P < 0.01). Furthermore, MBP was similar 
between the two kinds of recovery despite an increase in Z(0) during passive 
compared to active recovery. These results suggest that the faster haemodynamic 
recovery towards baseline and the decrease in cardiac preload during passive 
recovery may be successfully prevented by cardiovascular regulatory mechanisms 
which include an increase in SVR, thus avoiding a drop in blood pressure.


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